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Ontdek Huidziekten met de Woods Lamp - Een Gids voor Diagnostiek en Behandeling

Woods lamp huidziekten

JOHN W. ELY, MD, MSPH, SANDRA ROSENFELD, MD, AND MARY SEABURY STONE, MD

Am Fam Physician. 2014,90(10):702-711

Author disclosure: No relevant financial affiliations.

Tinea infections are caused by dermatophytes and are classified by the involved site. The most common infections in prepubertal children are tinea corporis and tinea capitis, whereas adolescents and adults are more likely to develop tinea cruris, tinea pedis, and tinea unguium (onychomycosis). The clinical diagnosis can be unreliable because tinea infections have many mimics, which can manifest identical lesions. For example, tinea corporis can be confused with eczema, tinea capitis can be confused with alopecia areata, and onychomycosis can be confused with dystrophic toe-nails from repeated low-level trauma. Physicians should confirm suspected onychomycosis and tinea capitis with a potassium hydroxide preparation or culture. Tinea corporis, tinea cruris, and tinea pedis generally respond to inexpensive topical agents such as terbinafine cream or butenafine cream, but oral antifungal agents may be indicated for extensive disease, failed topical treatment, immunocompromised patients, or severe moccasin-type tinea pedis. Oral terbinafine is first-line therapy for tinea capitis and onychomycosis because of its tolerability, high cure rate, and low cost. However, kerion should be treated with griseofulvin unless Trichophyton has been documented as the pathogen. Failure to treat kerion promptly can lead to scarring and permanent hair loss.

Clinical recommendationEvidence ratingReferences
Tinea corporis, tinea cruris, and tinea pedis can often be diagnosed based on appearance, but a potassium hydroxide preparation or culture should be performed when the appearance is atypical.C 2
Acceptable treatments for tinea capitis, with shorter treatment courses than griseofulvin, include terbinafine (Lamisil) and fluconazole (Diflucan).A 14 – 16
The diagnosis of onychomycosis should generally be confirmed with a test such as potassium hydroxide preparation, culture, or periodic acid–Schiff stain before initiating treatment.C 27

Gram-Negative Toe Web Infections

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (BY-NC-4.0), https://creativecommons.org/licenses/by-nc/4.0/, which permits unrestricted noncommercial use, distribution, and reproduction in any medium, provided the original authors and source are credited.

Gram-negative bacterial toe web infection (GNTWI) is a common dermatologic condition affecting the interdigital spaces. The clinical presentation ranges from mild erythema to exudative maceration of the web spaces that may extend to in other areas of the foot and lead to cellulitis in severe cases. Pseudomonas aeruginosa is the most commonly identified etiologic agent. Occlusive and humid environments, pre-existing dermatologic conditions, and fungal infections increase the risk of developing GNTWI. GNTWI has a broad differential diagnosis including erythrasma, tinea pedis, pitted keratolysis, eczematous dermatitis, and malignancies. Diagnosis is performed using bacterial and fungal cultures. There is a lack of a standardized treatment regimen for GNTWI. While GNTWI is fairly common, it may still be under-recognized by dermatologists due to the limited medical literature. This article presents a review of GNTWI, its clinical features, epidemiologic factors, etiologic agents, predisposing factors, diagnostic methods, and therapeutic options.

Keywords: Gram-negative bacteria, toe web infection, toe web intertrigo, Pseudomonas

Enhancing Healthcare Team Outcomes

The diagnosis and the treatment of HI are interprofessional including dermatologists, neurologists, pediatricians, orthopedic surgeons, ophthalmologists, dentists, and geneticists. Since the treatment of skin lesions is not necessary and has an excellent prognosis, the focus must be on the management of noncutaneous abnormalities with regular follow-up by a dermatologist and specialty-trained dermatology nurse providing coordination of care. [Level V]

Kouzak SS, Mendes MS, Costa IM. Cutaneous mosaicisms: concepts, patterns and classifications. An Bras Dermatol. 2013 Jul-Aug, 88 (4):507-17. [PMC free article : PMC3760924 ] [PubMed : 24068120 ]

Glover MT, Brett EM, Atherton DJ. Hypomelanosis of Ito: spectrum of the disease. J Pediatr. 1989 Jul, 115 (1):75-80. [PubMed : 2738798 ]

Pascual-Castroviejo I, Roche C, Martinez-Bermejo A, Arcas J, Lopez-Martin V, Tendero A, Esquiroz JL, Pascual-Pascual SI. Hypomelanosis of ITO. A study of 76 infantile cases. Brain Dev. 1998 Jan, 20 (1):36-43. [PubMed : 9533559 ]

Barbel P, Brown S, Peterson K. Identification of Hypomelanosis of Ito in Pediatric Primary Care. J Pediatr Health Care. 2015 Nov-Dec, 29 (6):551-4. [PubMed : 25698313 ]

Ardinger HH, Bell WE. Hypomelanosis of Ito. Wood's light and magnetic resonance imaging as diagnostic measures. Arch Neurol. 1986 Aug, 43 (8):848-50. [PubMed : 3729768 ]

Ruiz-Maldonado R, Toussaint S, Tamayo L, Laterza A, del Castillo V. Hypomelanosis of Ito: diagnostic criteria and report of 41 cases. Pediatr Dermatol. 1992 Mar, 9 (1):1-10. [PubMed : 1574469 ]

Disclosure: Amal Chamli declares no relevant financial relationships with ineligible companies.

Disclosure: Noureddine Litaiem declares no relevant financial relationships with ineligible companies.

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